Max in WT synaptoneurosomes, suggesting that Src signaling may be downregulated in KI synapses. On the flip side, our power to rescue SERT purpose in KI midbrain synaptoneurosomes because of the inhibition of FAK implies elevated FAK signaling downstream with the Pro32Pro33 mutant, as confirmed by enhanced pFAK localization in https://pro33login00009.liberty-blog.com/32409811/5-tips-about-pro33-you-can-use-today